BioOncology Watch

Timely Information for Practicing Physicians

 

september 2002

 

RITUXIMAB-REFRACTORY FOLLICULAR NON-HODGKIN'S LYMPHOMA (NHL)

Radioimmunotherapy with ibritumomab tiuxetan (Zevalin; IDEC Pharmaceuticals). Thomas Witzig and coworkers administered radiolabeled ibritumomab tiuxetan on an outpatient basis to 54 patients with follicular NHL refractory to rituximab (patients with no objective response to rituximab 375 mg/m2 weekly for 4 weeks or patients that had relapsed within 6 months after rituximab treatment).  Patients received rituximab 250 mg/m2 intravenously (IV) on Days 1 and 8 to delete peripheral blood B cells followed by yttrium-90 ibritumomab tiuxetan 0.4 mCi/kg IV on Day 8.  The response rate was 74% (15% complete response rate).  The Kaplan-Meier-estimated time to progression was 6.8 months (range, 1.1 to ³ 25.9 months) for all patients and 8.7 months for responders.  The most common adverse events were hematologic and the incidence of grade 4 neutropenia correlated with increasing bone marrow involvement with NHL (p = 0.004).  The percentage of patients that had grade 4 neutropenia, thrombocytopenia, or anemia was 35%, 9%, and 4%, respectively. This study shows that ibritumomab tiuxetan treatment can achieve high response rates in patients with follicular NHL refractory to rituximab, but the duration of response is generally short-lived.  (Witzig TE, et al. Blood 2002;100:3262-3269)

 

CHRONIC LYMPHOCYTIC LEUKEMIA (CLL)

First-line treatment with Campath-1H.  Jeanette Lundin and associates conducted a multicenter phase II study in which 41 patients with previously untreated B-CLL were given the humanized anti-CD52 monoclonal antibody, Campath-1H subcutaneously 3 x weekly for up to 18 weeks.  Twenty-five patients (61%) completed all 18 weeks of treatment.  Thirty-eight patients were evaluable and the overall response rate was 87% (95% CI, 76% to 98%; 19% complete remissions). CLL cells were cleared from blood in 95% of patients (in a median of 21 days).  The median time to treatment failure has not been reached (range, 8 to 44+ months).  The incidence of grade 4 neutropenia was 21% and 10% of patients developed CMV reactivation (all patients rapidly responded to IV ganciclovir). Transient injection site skin reactions occurred in 90% of patients, however, first-dose reactions commonly experienced following intravenous Campath-1H were rare or absent. Thus, subcutaneous Campath-1H resulted in few first-dose adverse events and may reduce health care costs compared to intravenous administration. (Lunden J, et al. Blood 2002;100:768-773)

 

Clinical significance of tumor necrosis factor-a (TNF-a) plasma level.  Alessandra Ferrajoli and colleagues determined TNF-a levels in peripheral blood samples obtained from 150 consecutive CLL patients.  The mean TNF-a plasma concentration was higher in CLL patients than in 20 healthy normal volunteers (16.4 vs. 8.7 pg/mL; p <0.0001).  In addition, higher TNF-a levels in CLL patients were associated with more advanced stage of disease, chromosomal abnormalities, higher serum beta-2 microglobulin levels, a higher percentage of cells expressing CD38, low hemoglobin levels, and thrombocytopenia.  Patients having a TNF-a plasma level >14 pg/mL had shorter survival times (p = 0.00001) and a Cox multivariate analysis identified the plasma TNF-a level to be an independent predictor of survival (p = 0.005).  These data indicate that the TNF-a plasma level is a prognostic factor for patients with CLL and that TNF-a may be a useful therapeutic target.  (Ferrajoli A, et al. Blood 2002;100:1215-1219)

 

MYELOID LEUKEMIA

Targeted a particle radioimmunotherapy.  Joseph Jurcic et al. conducted a phase I study of HuM195, a humanized anti-CD33 monoclonal antibody, labeled with 213Bi in patients with advanced myeloid leukemia. The a-emitters can selectively kill cancer cells with a single atomic decay and may avoid the prolonged myelosuppression associated with standard b particle-emitting isotope therapy.  Eighteen patients were treated with 10.36 to 37.0 MBq/kg 213Bi-HuM195 and no significant extramedullary toxicity was seen.  Myelosuppression occurred in all 17 evaluable patients with a median time to recovery of 22 days.  The absorbed dose ratios between leukemic sites and whole body were 1000-fold greater than those seen with b-emitters.  A reduction in the percentage of bone marrow blasts was achieved in 14 (78%) of 18 patients.  This study demonstrated that targeted a particle immunotherapy in humans is feasible.  (Jurcic JG, et al. Blood 2002;100:1233-1239)

 

MULTIPLE MYELOMA (MM)

Thalidomide and doxorubicin and deep venous thrombosis (DVT).  Thalidomide has minimal prothrombogenic activity when used as single-agent therapy.  However, the combination of thalidomide with chemotherapy has been reported to be associated with an increased risk of DVT.  Maurizio Zangari and colleagues retrospectively analyzed the incidence of DVT in 232 MM patients treated in two protocols investigating the combination of thalidomide treatments with chemotherapy.  The patients in these protocols were balanced for known risk factors for DVT. They found that patients receiving thalidomide plus DT-PACE (a doxorubicin-containing regimen consisting of dexamethasone, thalidomide, cisplatin, doxorubicin, cyclophosphamide, and etoposide) had a greater incidence of DVT than those treated with thalidomide plus DCEP-T (a non-doxorubicin-containing regimen consisting of dexamethasone, cyclophosphamide, etoposide, cisplatin, and thalidomide) (31of 192 patients [16%] vs. 1 of 40 patients [2.5%]; p= 0.02).  Of the 32 patients with DVT, 11 patients developed a thrombosis at the site of a central venous catheter and 21 patients developed DVT at distant sites.  One of these patients also had a nonfatal pulmonary embolism.  These data suggest that MM patients treated with thalidomide and doxorubicin have an increased risk for DVT.  (Zangari M, et al. Blood 2002;100:1168-1171) 

 

 

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